All viruses bind to their hosts and introduce their genetic material into the host cell as part of their replication cycle.

In addition, the more known about how and where viruses and bacteria bind to cells, the better the chances of designing drugs to interfere with the process.

The viruses bind to the antigen binding site at the other end of the antibody.

It does this by attachment - or adsorption --- onto a susceptible cell; a cell which holds a receptor that the virus can bind to.

The virus binds to the receptor, then fuses to the cell, and fusion allows HIV to work its way inside.

Researchers "found that the mutations at two places in the gene, identified as 182 and 192, allow the virus to bind to both bird and human receptors."

"The virus then binds to the decoy instead of penetrating the cell."

The virus binds to specific receptors on cells and the core particle enters the cell cytoplasm.

The modified virus would go off and bind to the cells in the body that HIV normally binds to.

It has been proposed that the viruses bind via these proteins and are then taken into the insect cell by receptor-mediated endocytosis.