This provides a means of delivering materials to the cells, intercellular communication, as well as removal of metabolic waste.

Currently, there are no proven mechanisms by which microvesicles trigger intercellular communication.

Biochemical evidence supports the idea that exRNA uptake is a common process, suggesting new pathways for intercellular communication.

These findings suggest that loss of intercellular communication via gap junctions is associated with gastric ulcer formation.

Most cells continue their vital activities, such as differentiation and proliferation, while maintaining homeostasis through intercellular communication via gap junctions.

These results suggest that the loss of intercellular communication mediated by gap junctions may be associated with the recurrence of gastric ulcers.

Possible mechanisms by which microvesicles trigger intercellular communication are paracrine, fusion and phagocytosis.

Cytokines are signals used in the immune system for intercellular communication.

N-Acetylgalactosamine is necessary for intercellular communication, and is concentrated in sensory nerve structures of both humans and animals.

They come from separate paths with respect to these systems of intercellular communication.