The observation lead to hypothesis that excess potassium in extracellular space is "siphoned" by the Muller cells to the vitreous humor.

Aldosterone, in turn, causes the body to rid itself of the excess potassium.

In normal people, taking potassium supplements or potassium-containing drugs is of no consequences, because the kidneys efficiently dispose of excess potassium.

In humans, mainly the kidneys are responsible for the regulation of serum potassium levels by excreting excess potassium via the urine.

As a result, they have developed this lateral nasal gland to supplement renal salt secretion by expelling excess potassium and sodium chloride.

RRT removes excess potassium, acid and phosphate that accumulate when the kidneys are unable to function normally and is required until kidney function is regained.

Low aldosterone levels may lead to salt loss and hyperkalemia (excess potassium).

Early humans evolved to conserve sodium, which was hard to obtain, and to excrete excess potassium, abundant in many fruits and vegetables.

In this setting, the human kidney developed a highly efficient capacity to excrete excess potassium.

But his death certificate listed the cause of death as an abnormal heart rhythm and excess potassium - two well-known results of digoxin poisoning - without ever mentioning the drug.